At the Cross-Roads of Blood Circulation, Is the Cardio-Vascular System Hit Every Time We Vaccinate?

The IV-induced bolus of vax particles systematically goes through the cardiovascular crossroad: the right heart chambers, the lungs, the left heart chambers, the aorta...always for every vaccinated!

I have two brothers and one sister.

My elder brother recently discovered his aorta had necrotized (likely a result of the vaccines and white clots). My older sister, who I had begged not to take the vaccines, didn’t listen like everybody else and now has atherosclerosis. Two out of three were harmed in the cardiovascular system…

I can’t imagine asking my younger brother about his health.

Either my family is cursed (possible), or the Covid vaccines - and vaccines in general - are truly harmful to the cardiovascular system (the two are not mutually exclusive sadly)…

It’s always dubious to generalize from anecdotes, but when there’s a convincing rationale, it’s harder to discount reality…

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Today I want to emphasize a central point of the Bolus Theory: no one knows where the bolus of particles injected directly into venules of the deltoid will end up, often it hits innocuous areas like muscles or fat.

WHAT WE CAN BE CERTAIN: THE BOLUS SYSTEMATICALLY PASSES THROUGH THE RIGH ATRIUM AND VENTRICLE, THE LUNGS, THE LEFT ATRIUM AND VENTRICLE, THE AORTA, AND AT LEAST ONE MAJOR ARTERY where it can transfect the linings, trigger concentrated T-cell attacks, and considerable endothelial damage.

That is a scary certainty:

100% of vaccine nanoparticles going IV will follow that cardiovascular passage!

Many have overly focused on myocarditis as being the hallmark of vaccine injuries, but myocarditis events are proportional to myocardial capillary exposure and relatively small: think 180 grams versus a 70kg body (70,000 grams). The brain, the liver, the intestines, the muscle, and the skin all have much larger endothelial surfaces and thus are more exposed.

However, the linings and the valves of the heart, the capillaries of the lungs, and the pulmonary arteries and veins, as well as the aorta, are systematically vulnerable to the vaccine particles that necessarily pass through them. That is a fundamental difference.

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A Longer Bolus

My calculation of the bolus to date has been based on the most intense bolus at the moment of injection. The closer the cut vessel is to the needle’s tip, the longer the instantaneous bolus. The bigger the diameter of the cut vessel the larger the bolus.

Let me try an analogy here. Say two whales are trying to capture a school of krill:

1. one smaller whale has a smaller gape - a smaller mouth (representing a smaller cut vessel in taking directly vaccine particles),

2. the other a much larger whale with a much larger gape (representing a larger cut vessel).

The closer the whales are to the school before they start evading and scrambling away in all directions, the larger the proportion of the school will be captured by a whale. It’s the same with the vaccine particles captured by pressure differential at the moment of injection. The closer to the point of expansion of the vaccine dose - the tip of the needle - the more particles will be captured by the aperture in the blood vessel as the vaccine dose expands

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Evidently, a larger whale with a larger gape will capture proportionally more krills based on the surface of its gape, playing the role of a fishing net. A gape twice as large will capture four times more krills at the same distance, supposing the krills would evade in all directions, including the whale’s mouth (which isn’t realistic, I know).

My visualization of the bolus had stopped there, as if the rest of the dose would go via the lymphatic system. That was likely a mistake on my part.

The lymphatic system, given its mass, will easily contain the pressure of the 0.3-0.5 ml push; in other words, the pressure differential between the muscle interstitial fluid and the cut blood vessel will remain high as long as the excess liquid isn’t completely evacuated. Given that the lymphatic system is very slow to move, that can take some time. That observation means that the muscle structure continues to pressure on the interstitial fluid, which will continue leaking directly into the bloodstream.

Coagulation of the severed blood vessels can’t happen as long as the blood pressure coming from the bloodstream is smaller than the interstitial fluid pressure. As long as the blood can’t penetrate, the leak can’t be stopped.

So the gap is still open, and the vaccine particles will continue being rushed through to the vascular system like sci-fi movie villains being sucked into outer space when a sealed door breaks and pressurized air is precipitated in the outer space vacuum.

The muscle pressure acts slower than the first bolus impulse, like a saline bag leaking the vaccine dose directly, faster or slower depending on the throughput permitted by the diameters of the severed vessels and the progressively falling pressure differential.

The example of 6-year old Milo Edberg vaccinated directly via a blood port showed how that can be dangerous with lungs issues, myocarditis, Guillain-Barré…

There comes a point when the throughput is tiny, and the dose is instantly diluted, making it innocuous. However, that means that a significant portion of the vaccine dose is actually injected directly into the bloodstream at high concentrations. If I recall well the Acuitas study on mice, 48% of the dose had leaked directly into the bloodstream within one hour. For Pfizer, we are talking roughly 5 billion nanoparticles injected directly into the bloodstream within an hour, and 20 billion for Moderna. All passing through the cardiovascular crossroads I mentioned above.

Even if, at first, the protective PolyEthylene Glycol coating hinders vaccine particles from penetrating endothelial linings when they are pushed by the blood flow, it is quite obvious that the coating gets eroded quite quickly. And even if, at any given moment, 99% of the nanoparticles are away from the linings, the flow mixing the blood would precipitate the particles against these walls at one moment or another.

This is frightening because if only 1‰ transfects, we are talking 5 to 20 million concentrated transfected cells within an hour, or 65 to 260 square centimeters of endothelium at risk troughout the body, but principally in the cardiovascular crossroad..

A rapid search on VAERS shows 20,293 adverse events related to the lungs!
Remember the under-reporting is very high, and some take years to materialize, given the pressure hospitals it’s possible only 1 in 100 got declared, that would mean 2 million lungs damaged, and I presume that wouldn’t account for asthma-type adverse reactions or long-term white clot clogging, both of which would have to be much higher.

Given activation by PEG erosion mostly happens in the lungs, it means that there should be more damage done to the left heart chambers, notably the left mitral and aortic valves, but given how busy and constrained cardiologists are, it would be surprising if 1 in 1,000 accident is reported.

• Atrial thrombosis 47

• Aortic thrombosis 102

• Cardiac ventricular thrombosis 103

• Arterial thrombosis 168

• Coronary thrombosis 239

• Deep vein thrombosis 9,822

• Pulmonary thrombosis 1,229

• Pulmonary necrosis 8
  (What’s the under-reporting factor here when I personally know 2?! Gilles and Laurent are in my 500 closest relations, that would translate to about 22 million people with necrotized lung nodes throughout the world)

• Asthma 5,047

• 710 aortic, arterial, or venous dissections.
  That’s a medium-term occurrence that is likely not easy to connect to vaccines.

The data shared by Major Tom Havilland is that embalmers are being hindered by white clots in 20% of the deceased they process. Those would be the most severe cases, meaning they didn’t necessarily find them all.

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I remember reading an article on heart transplants in the US where they had found 85% of the deceased they were taking the hearts from had arteriosclerosis.

This highlights the considerable level of damage vaccine boluses - before COVID vaccines - had already inflicted on the population. We are indeed running to the abyss, but thankfully there’s hope for many.

Those whose lungs have been necrotized can improve and possibly save the rest of their lungs from further damage, but it will be very difficult to bring back their lost pulmonary capacity. However, there’s hope for those with damaged arteries with white clots and calcified linings, if they take their destiny in hands and start helping their body finish the repair.

As I have stated multiple times, the natural course of repair is (1) coagulation (happens at the time of immune attacks), (2) cellular repair, (3) revascularization, and ultimately when all is repaired and revascularised, (4) dissolve the natural bandage (fibrin).

If the body isn’t able to do it alone, revascularisation and endothelial repair can be obtained by stem cell stimulation through hyperoxygenation of the blood. There are a variety of ways to increase the presence of O2 in the blood. Many readers have pointed to chlorine dioxide or ozone¹ with satisfactory results. Personally, I believe natural oxygenation via our lungs is preferable, as those were specifically designed for gas-blood exchanges. The repair stimulation might take a month or two, and then a high-dose fibrinolytic treatment can begin to liberate the cardio-vascular system of the fibrin. (see “The Needle’s Secret” for more details).

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In the coming days, I will publish a brief article on the number and duration of OT sessions, notably if you use an oxygen concentrator, to repair the damage done. Many of you have been asking questions, and I will do my best to provide you with some guidance.

Looking forward to your comments.
Feel free to order “The Needle’s Secret” as a Christmas present. I couldn’t have dreamt of better reviews.

Please consider supporting my family, or becoming a paid subscriber.

A big hug from Paris. Love.

Marc

My identifier on PayPal is marc.girardot@icloud.com

1. “Oxygen-Ozone (O2-O3) Therapy in Peripheral Arterial Disease (PAD): A Review Study” by Juchniewicz et al. - Reference